Food Allergies
This is a concerning reality that I have to deal with every day.
Food Allergies
Though estimates also vary widely for food intolerance, a condition in which the body is unable to produce enough of the natural digestive chemicals to break down a particular type of food, the FDA estimates that approximately 28 per cent of Americans suffer from some form of this condition. About 8 percent of children 6 years and younger have food intolerances. Of this group, between 2 percent and 4 percent may have allergic reactions to food. An estimated 1 percent to 2 percent of adults are sensitive to food or food additives. All allergies, whether they are caused by grasses or household dust, affect as many as 40 to 50 million people in the United States.
The development of allergic diseases seems to involve complex interactions between genetic factors and diet and other environmental factors.
We must consider the genetic diversity of the human race when conducting research and development in this area.
The effects range from mild inconvenience to life threatening. One bite can be deadly
While food allergies now lead to 30,000 emergency-room visits a year in this country, the number of fatalities is relatively low—between 150 and 200 annually. A horse is likelier to kill you in the United States than all the peanuts, shrimp, and gluten combined. So why the alarm? No doubt the vivid particulars of a serious allergic reaction—constricted airways, rapid pulse, and loss of consciousness—grip our imagination. What’s more, the triggers seem ubiquitous. Severe anaphylactic shock can be brought on by shared utensils at a buffet, a single careless waiter, residue on a doorknob, trace amounts of peanut dust in a ventilation system, and, yes, kissing. The numbers could be higher than that. Hospitals may not be report all allergic reactions they treat or some people may not come to the emergency room or see a doctor when an attack happens.
Since more children have allergies, there are more kids at risk for severe allergic reactions.
More and more children have severe food allergies. But no one knows just how many more there are.
The two most common culprits are lactose (found in milk-based products) and gluten (found in wheat-based products). More than 170 foods have been identified as allergens, including fruits, sesame seeds, sunflower seeds, poppy seeds, mollusks, peas, lentils, and beans other than green beans. Eight foods account for 90 percent of severe allergies - peanuts, tree nuts (walnuts, pecans, etc.), fish, shellfish, eggs, milk, soy and wheat. The other cases include latex, insect venom and reactions to medications. Peanuts are the leading cause of allergic reactions. Peanut and tree nut allergies affect about 3 million Americans.
Facts gathered from other web pages:
- Food allergies in Australian children have risen 12-fold since 1995
- In addition, numerous studies attest to the dramatic rise in food allergies in the United States. In fact, the number of children who have food allergies has quadrupled over the last few decades, and the Journal of Allergy and Clinical Immunology reports that the number of children allergic to peanuts increased two-fold over a single five-year period from 1997 to 2002.
- It’s estimated that as many as 7 million Americans suffer from allergies to common foods, and as many as 200 people die every year from allergic reactions. While no one is quite sure what causes food allergies, the numbers are on the rise.
- NIH the leading researcher on allergies - estimates that Some 12 million Americans, including about 2 to 4 percent of adults and 6 to 8 percent of children under four, have a true food allergy and one out of four allergic individuals will have an "accidental exposure that leads to a food-induced reaction even with assiduous avoidance of known food allergens," according to findings revealed in a report published in June by a panel convened at the request of Congress. The panel consisted of national and international food allergy experts brought together by the NIH on behalf of the Secretary of Health and Human Services (HHS).
- Roughly 2.2 million kids in the U.S. have a life-threatening food allergy, A mere molecule of the offending protein is enough to send an allergic kid into anaphylactic shock, which can result in death.
- The number of food-allergy sufferers in America has risen from 7 million to 12 million over the last five years, according to the nonprofit Food Allergy Initiative. As with many medical statistics, there’s some debate over how much this increase simply reflects a growing ability to spot the problem. Still, researchers at the Mount Sinai School of Medicine found that 1 in 250 preschoolers suffered from peanut allergy—the most dangerous variety—in 1997; five years later, that number had jumped to 1 in 125.
Low Folate Could Contribute to Allergies
Low Folate Could Contribute to Allergies
"...a recent report from the National Health and Nutrition Examination Survey (NHANES), published in the Journal of Allergy and Clinical Immunology...
Folate linked to less wheezing
...the people with the highest folate levels had the lowest levels of total and specific allergy-related antibodies and were the least likely to have wheezing.
Nutrition for allergy prevention
Folate, part of the B vitamin-complex is found abundantly in nature. Folate-rich foods include whole grains, vegetables, legumes, and some fruits like avocados and strawberries. Folate plays a critical role in cell division, and its importance to the developing fetus came to light in the 1970s when congenital neural tube defects and cleft palate were linked to low levels during pregnancy. Efforts to increase intake through education and fortification programs have helped improve folate status in many parts of the world; nevertheless, it remains one of the most common vitamin deficiencies worldwide.
More recent research has shed light on folate’s importance in modulating the immune system and preventing inflammatory processes that can lead to diseases such as cardiovascular disease and rheumatoid arthritis. Although it only provides preliminary evidence, the new report from NHANES could lead to a broader understanding of the importance of folate-rich foods in disease prevention. “[Our] findings suggest that dietary folic acid and factors regulating its metabolism might play an important role in the development and perpetuation of allergy and asthma,” the study’s authors said.
Folate, part of the B vitamin-complex is found abundantly in nature. Folate-rich foods include whole grains, vegetables, legumes, and some fruits like avocados and strawberries. Folate plays a critical role in cell division, and its importance to the developing fetus came to light in the 1970s when congenital neural tube defects and cleft palate were linked to low levels during pregnancy. Efforts to increase intake through education and fortification programs have helped improve folate status in many parts of the world; nevertheless, it remains one of the most common vitamin deficiencies worldwide.
More recent research has shed light on folate’s importance in modulating the immune system and preventing inflammatory processes that can lead to diseases such as cardiovascular disease and rheumatoid arthritis. Although it only provides preliminary evidence, the new report from NHANES could lead to a broader understanding of the importance of folate-rich foods in disease prevention. “[Our] findings suggest that dietary folic acid and factors regulating its metabolism might play an important role in the development and perpetuation of allergy and asthma,” the study’s authors said.
Getting enough folate
With so much evidence pointing toward folate’s importance in preventing chronic disease, it’s a good idea to evaluate your diet and be sure you are getting enough.
• Eat leafy greens every day. Try spinach, asparagus, and collard greens. Dark green lettuces can also be good sources, especially because they are not heated—some folate is lost during cooking.
• Choose whole grain breakfast cereals or cereals that are fortified with folic acid.
• Include lentils and beans, like pintos, black, and garbanzo beans, in soups and on salads, or try beans, brown rice, and avocado for a main dish.
• Consider a multivitamin. Folic acid, the form of folate used in supplements, is very effective for improving folate status.
(J Allergy Clin Immunol 2009;123:1253–9)"
Oral Allergy Syndrome on the rise
“Fresh fruit, vegetables and nuts are common causes and foods that are more likely to trigger it include celery, carrots, tomatoes, apples, peaches, pears and hazelnuts.
... This means that OAS is likely to be more prevalent than peanut allergies, which are far less common, according to Allergy UK spokeswoman Lindsey McManus.
... She told FoodNavigator.com: “It may well be that because the incidence of hay fever is very high, it (OAS) is probably going to be higher than peanut allergies.
“We haven’t got any statistics but we are seeing more of it across the board (among adults and children).”
... Also according to the BBC, records indicate that in a six year period, the rate of final clinically diagnosed cases of OAS rose from about one per 100,000 of the population in the Cardiff and South Wales area, to five in the year 2007/8.
However the reason for such increases could be improved diagnostic procedures. “
The article mentions that the syndrome is "linked" to hay fever... which affects every 10 people in the UK. But I'm wondering about this correlation:
| OAS Foods | Rank on Full List (47) | Score | |
| Peaches | Peach | 1 | 100 |
| Apples | Apple | 2 | 93 |
| . | Bell Pepper | 3 | 83 |
| Celery | Celery | 4 | 82 |
| . | Nectarine | 5 | 81 |
| . | Strawberries | 6 | 80 |
| . | Cherries | 7 | 73 |
| . | Kale | 8 | 69 |
| . | Lettuce | 9 | 67 |
| . | Grapes (imported) | 10 | 66 |
| Carrots | Carrot | 11 | 63 |
| Pears | Pear | 12 | 63 |
| Tomatoes | . | 34 | 29 |
| Hazelnuts | . | (not tested) | N/A |
5 of the 7 fruits and vegetables listed as likely to trigger OAS are on EWG's "Dirty Dozen" of foods with high pesticide loads.
Tomatoes, though they carry a pesticide load are in the mid range as compared to the Dirty Dozen and the "Clean 15".
To quote the methodology used by this activist group:
“Consistent with two previous EWG investigations, fruits topped the list of the consistently most contaminated fruits and vegetables, with seven of the 12 most contaminated foods. The seven were peaches leading the list, then apples, nectarines and strawberries, cherries, and imported grapes, and pears. Among these seven fruits:
· Nectarines had the highest percentage of samples test positive for pesticides (97.3 percent), followed by peaches (96.7 percent) and apples (94.1 percent).
· Peaches had the highest likelihood of multiple pesticides on a single sample - 87.0 percent had two or more pesticide residues — followed by nectarines (85.3 percent) and apples (82.3 percent).
· Peaches and apples had the most pesticides detected on a single sample, with nine pesticides on a single sample, followed by strawberries and imported grapes where eight pesticides were found on a single sample of each fruit.
· Peaches had the most pesticides overall, with some combination of up to 53 pesticides found on the samples tested, followed by apples with 50 pesticides and strawberries with 38.
Sweet bell peppers, celery, kale, lettuce, and carrots are the vegetables most likely to expose consumers to pesticides. Among these five vegetables:
· Celery had the highest of percentage of samples test positive for pesticides (94.1 percent), followed by sweet bell peppers (81.5 percent) and carrots (82.3 percent).
· Celery also had the highest likelihood of multiple pesticides on a single vegetable (79.8 percent of samples), followed by sweet bell peppers (62.2 percent) and kale (53.1 percent).
· Sweet bell peppers had the most pesticides detected on a single sample (11 found on one sample), followed by kale (10 found on one sample), then lettuce and celery (both with nine).
· Sweet bell peppers were the vegetable with the most pesticides overall, with 64, followed by lettuce with 57 and carrots with 40.”
I wonder if this will effect the incidence of allergies...
More pesticide residue allowed on citrus, rules EPA (click link for full article)
"The Environmental Protection Agency (EPA) has increased the tolerated level of pesticide residue for citrus fruits and oils, following a petition from Dow AgroSciences.
The EPA conducts random tests for residues of the insecticide methoxyfenozide at the grower, food manufacture and retail level. If they are found to be too high, ingredients can be withdrawn and growers face fines as well as the loss of income from crops that may have cost them thousands of dollars to produce. Although food manufacturers are not held responsible for ensuring that pesticide levels are acceptable, they are susceptible to disruption if the EPA withdraws an ingredient found to have excessive pesticide residues.
The agency said that as a result of the petition from Dow AgroSciences, which produces the insecticide, it has reviewed the available scientific data for methoxyfenozide, and has increased the maximum tolerance level for citrus fruit from two parts per million (ppm) to ten ppm, and the tolerance for citrus oil from 70 to 100ppm.
In its final rule, it said that the pesticide is “not acutely toxic” and that studies show “it has few or no biologically significant toxic effects at relatively low-dose levels in many animal studies and only mild or no toxic effects at relatively high-dose levels.”
No comments were received in response to the filing of the petition prepared by Dow AgroSciences. Objections to the rule or requests for a hearing can be filed prior to November 2.
Further information on the EPA’s final rule can be found online here . "
Aflotoxin and tree nuts
EFSA clears raising permitted aflatoxin levels in tree nuts by 150 per cent
I have to wonder if any of this has an impact as well.
This was reported 7 Jul 2009 by NutraIngredients.com
"Public health would not be harmed if the maximum level of aflatoxins permitted in all tree nuts was increased by 150 per cent, the European Food Safety Authority (EFSA) has said.
The European food safety watchdog’s Scientific Panel on Contaminants in the Food Chain (CONTAM) made the announcement as it recommended raising maximum levels of the toxic mould from 4µ/kg to 10µg/kg for all tree nuts.Aflatoxins can occur in food and feed as a result of fungal contamination by moulds, primarily by Aspergillus flavus and A. parasiticus under warm and humid conditions and are most likely to be present in tree nuts.
However, even as the panel adopted the opinion, it stressed that “that exposure to aflatoxins from all sources should be as low as reasonably achievable, because aflatoxins are genotoxic and carcinogenic, and that priority should be given to reducing the numbers of highly contaminated foods reaching the market, irrespective of the commodity involved”.
Minor impact
EFSA was originally tasked with re-examining the levels by the European Commission in January 2007. This followed a proposal from the Codex Committee for Food Additives and Contaminants (CCFAC) to the bloc to increase aflatoxins limits in almonds, hazelnuts and pistachios. At that time the panel said changing the level from 4µ/kg to 8 or 10 µg/kg for these three types of nuts would “have a minor impact on the estimates of dietary exposure, cancer risk and the calculated margin of exposures”.
The previous estimates showed increasing the maximum levels for total aflatoxins in almonds, hazelnuts and pistachios from 4 to 8 or 10 µg/kg would increase total dietary exposure to the substance by around one per cent. By including all other thee nuts, the panel noted that only about 0.5 per cent of such nuts would have concentrations in this range except for Brazil nuts with 2.4 per cent.
...
The panel reached its latest conclusion after looking at almost 35,000 incidents of aflatoxin-tainted food products - including tree nuts other than almonds, hazelnuts and pistachios - in 2006.
Short deadline
However the panel appeared to admonish the Commission for not giving it sufficient time to gather more recent information.
“The short deadline of the Commission request for the current statement did not allow EFSA to issue a complementary call for further information, thus EFSA relied on existing information on aflatoxin in food collected in 2006," said an EFSA statement. "
This amounts to: "Let's lower the bar (and not think about it too much) because there's too many aflotoxin-tainted products".
Aflotoxin was the same stuff that poisoned and killed pets in 2006
Changes the bar will make precautions like this more difficult to implement:
"Although grain containing low levels of aflatoxin can be fed to livestock and poultry, risk is increased anytime aflatoxin is present at any concentration. If symptoms occur, discontinue feeding contaminated grain and return to an aflatoxin free diet immediately."
This doesn't just affect livestock:
Case-control study of an acute aflatoxicosis outbreak, Kenya, 2004.
"During January-June 2004, an aflatoxicosis outbreak in eastern Kenya resulted in 317 cases and 125 deaths."
From WrongDiagnosis.com
"Aflatoxins are toxins produced by fungus from the Aspergillus genus. The toxin is carcinogenic in humans and can also cause other health effect, particularly liver problems. "
There seems to be a connection with all of this and the toxic load that a person heaps onto their livers to "clean up" for them.
Add to this extra hormones from xenoestrogens etc., pollution of any kind, the waste products in the medicines that we take, the chemicals in our soaps and shampoos, makeup... It reminds me of a comedy movie I watched a long time ago, but in reality it's not so funny.
One study reports certain pesticides increase allergic reactions
In a study reported by Elsevier Science Ireland Ltd.
Various pesticides, especially phenthoate (PAP), chlornitrofen (CNP) and paraquat (PQ), increased histamine release.
"These results suggest that various pesticides as environmental pollutants exacerbate allergic diseases."
phenthoate (PAP) (information gathered from the link)
"Phenthoate is an organophosphorus pesticide of moderate toxicity. It
is readily absorbed through the intact skin, from the
gastrointestinal tract and by inhalation. Repeated exposure may have
a cumulative effect on cholinesterase levels."
Cholinesterase inhibition after metabolism. Orally administered
phenthoate is readily converted to phenthoate-oxon, which is the
active form, by liver microsomal enzymes.
Single dose oral toxicity was determined for rats, mice, guinea-pigs, rabbits, hares and dogs. Single dose dermal toxicity for rats.
Ironically, toxicity for mammals: repeated doses for oral, dermal, inhalation and cumulation have "no information".
Dietary studies for short-term effects - "no information"
There was an 88 week rat study to determine the highest non-toxic dose
Supplementary studies of toxicity - Carcinogenicity: No information; Teratosenicity: No information; Reproduction: No information; Neurotoxicity: No information.
Two microbial studies were run to determine that phenthoate is "non-mutagenic" for several strains of Salmonella typhimurium and Escherichia coli.
Toxicology on Man
2.2.2 Dangerous doses
Single: No information.
Repeated: No information.
2.2.3 Observations of occupationally exposed workers
Spraymen in a WHO supervised spray programme showed no ill-effects
over an extended period of spraying. Whole blood cholinesterase
levels were 90% of normal after three days of spraying (tintometric
method).
2.2.4 Observations on exposure of the general population
The population in villages undergoing WHO supervised spraying showed
no signs or symptoms for a 15-day observation period. No inhibition
of whole blood cholinesterase occurred (tintometric method).
2.2.5 Observations of volunteers
Young adult male military recruits wearing garments with sleeves
treated with 1% phenthoate for six weeks, 34-48 hours/week, showed
no noticeable ill-effects.
2.2.6 Reported mishaps
No information.
paraquat (PQ) (information gathered from the link)
Paraquat is one of the most widely used herbicides in the world. It has had a tarnished reputation because of its acute oral toxicity and ill-health associated with operators - particularly in the plantation sectors of many developing countries.
What is paraquat?
Paraquat is one of the most widely used herbicides, and held the largest share of the global herbicide market until recently overtaken by glyphosate. Paraquat is sold in about 130 countries for use on large and small farms, plantations and estates and in non-agricultural weed control. It is a quick acting, non-selective herbicide, which destroys green plant tissue on contact and by translocation within the plant.
The biggest markets for the company's paraquat products are the US, Japan, Malaysia, Thailand, Mexico, France and Brazil and annual sales are still increasing(3). For many years paraquat accounted for a big part of Zeneca's agro-chemical sales (24% in 1987(4) ) and is still a major product in the agrochemical portfolio.
Uses and usage
Paraquat is used to control broad-leaved weeds and grasses, being less effective on deep rooted plants such as dandelions. It does not harm mature bark, and is thus widely used for weed control in fruit orchards and plantation crops, including coffee, cocoa, coconut, oil palms, rubber, bananas, vines, olives and tea, ornamental trees and shrubs and in forestry. Other uses include weed control in alfalfa, onion, leeks, sugar beet, asparagus. It is used for weed control on non-crop land and can be used as a defoliant for cotton and hops before harvesting. Paraquat is used as a desiccant for pineapples, sugar cane, soya beans and sunflower(5). In pineapples, for example, paraquat is applied after harvest to accelerate the drying out process and enabling plants to be burnt after 3-5 weeks, compared to 13 weeks after the alternative cutting and natural drying.
Paraquat is increasingly used to destroy weeds in preparing land for planting in combination with no-till agricultural practices which minimise ploughing and help prevent soil erosion. Although toxic to fish, it is used as an aquatic herbicide where it is absorbed by plant matter and silt.
Toxic effects
Acute toxicity
Paraquat is highly toxic to animals and has serious and irreversible delayed effects if absorbed. As little as one teaspoonful of the active ingredient is fatal. Death occurs up to 30 days after ingestion. Absorbed paraquat is distributed via the bloodstream to practically all areas of the body. The lungs selectively accumulate paraquat, and therefore contain higher concentrations than other tissues. This develops into pulmonary oedema and other lung damage, leading to fibrosis. Liver damage occurs and renal failure may follow as the kidneys remove absorbed paraquat.
At spray strength paraquat is of relatively low acute toxicity. It is classified as a solid and with an acute oral LD50 for rats of 157 mg/kg, which puts it into WHO as Class II 'moderately hazardous'(6). Paraquat is also toxic if absorbed through the skin, see 'health issues' below. The minimum lethal dose by oral ingestion in human beings is about 35 mg/kg body weight, although less could be lethal without treatment. In dogs the lethal dose is 25-50 mg/kg and in cows and sheep 50-75 mg/kg(7). There is no vapour toxicity, but it can cause nose bleeding if inhaled.
No antidote for poisoning exists although it is recommended that the highly absorbent Fuller's Earth is administered. Hospital care must be sought without delay.
In occupational use the main route of exposure is through the skin and the worst cases of exposure occur during knapsack spraying. Continued exposure, as encountered by spray operators on plantations, is reported to affect the skin, eyes, nose and finger nails. Skin problems include mild irritation, blistering and ulceration, desquamation (peeling of the outer layer of the skin), necrosis (cell-death in skin tissue), dermatitis of the hands and in some cases scrotal areas (from leaking spray machines soaking trousers)(11). Severe exposure on hands has resulted in nail damage, ranging from localised discoloration to temporary nail loss(12). Eye splashes can result in irritation and inflamed eyelids (blepharitis) and visual acuity can decrease(13). A study in Thailand found clear indications of caustic burns on the feet after working with spinning disk applicators(14).
Offhand I couldn't find anything on chlornitrofen (CNP)
Breakthrough offers food allergy hope
This study is a start to figuring out how anaphylaxis works.
(excerpt)
Scottish scientists have reported a breakthrough that could slash the numbers of fatal cases of serious allergic reactions to peanuts and other foods.
According to findings published in the Proceedings of the National Association of Sciences (PNAS) a molecule called interleukin-33 amplifies allergic reactions. Researchers from the University of Glasgow also report a development of a biological agent to reduce the symptoms.
“Our study showed that IL-33 plays a pivotal role in hugely increasing the inflammation experienced during a period of anaphylactic shock and led us to understand how to intervene to reduce its impact.”
Using a mouse model, the researchers showed that blocking the IL-33 molecule could reduce the severity of the attack.
“This approach does not stop the allergic reaction altogether. It blocks the amplification of the reaction triggered by IL-33, not the allergic response itself.
“We are now further studying the role of IL-33 in anaphylaxis and similar disorders, and our plans are to further these studies on food, venoms and drugs-mediated anaphylaxis,” said Dr Melendez.
Source: PNAS
Volume 106, Number 24, Pages 9773-9778, doi: 10.1073/pnas.0901206106
"The cytokine interleukin-33 mediates anaphylactic shock"
Authors: P.N. Pushparaj, H.K. Tay, S.C. H'ng, N. Pitman, D. Xu, A. McKenzie, F.Y. Liew, A.J. Melendez
reported by Food Navigator
Allergy study shows progress for potential treatment
The article excerpt below shows a study where basic intestinal health measures - taking a prebiotic fiber to feed the beneficial bacteria in the intestines, and also supplementing with a particular probiotic bacteria - desensitized the mice to milk allergy.
I've read places that mention 80% of your immune system is in your intestines. Since allergies are an immune response this result seems to make sense. It'll be interesting to see what happens with the human studies.
A supplement combining a prebiotic fibre and a probiotic strain may reduce allergic responses to cow’s milk, when used in conjunction with small amounts of whey, says a new study.
“Dietary supplementation with [prebiotic] Immunofortis, [probiotic] Bifidobacterium breve M-16V, and particularly the synbiotic mixture, provided during sensitization, reduces the allergic effector response in a murine model of IgE-mediated hypersensitivity that mimics the human route of sensitization,” wrote the researchers led by Leon Knippels from Danone Research–Centre for Specialised Nutrition and the University of Utrecht.
Study details
Using a mouse model of orally induced cow’s milk allergy, the Netherlands-based researchers tested the effects of dietary supplementation with a prebiotic mixture (2 per cent, Immunofortis, a mix of galacto-oligosaccharides and fructo-oligosaccharides), a probiotic strain (2 per cent, B. breve M-16V, Morinaga Milk Industry), or a synbiotic (2 per cent) mix of both.
“Mice fed pro- and prebiotic diets had a significantly reduced acute allergic skin response upon whey challenge in the ear compared with whey-sensitized mice fed the control diet,” report the researchers. “The synbiotic diet was even more effective and almost completely prevented occurrence of the acute skin response as well as anaphylactic shock reactions.”
While IgE levels were not affected by any of the interventions, said the researchers, but IgG2a levels were – the antibody specific to whey. These increases in IgG2a may reflect a response by T-cells in the pro-, pre-, or synbiotic diet, said the researchers.
“Synbiotics comprise a promising concept that may be more effective in reducing allergic symptoms than single preparations of pre- or probiotics,” concluded the researchers.
Source: Journal of Nutrition
July 2009, Volume 139, Pages 1398-1403, doi:10.3945/jn.109.108514
“Cow Milk Allergy Symptoms Are Reduced in Mice Fed Dietary Synbiotics during Oral Sensitization with Whey”
Authors: B. Schouten, B.C.A.M. van Esch, G.A. Hofman, S.A.C.M. van Doorn, J. Knol, A.J. Nauta, J. Garssen, L.E.M. Willemsen, L.M.J. Knippels
China approves "allergy-battling" probiotics
Related news from NutraIngredients.com (click link for entire article):
China approves allergy-battling probiotics
According to GenMont, the approvals for its strains GM-080 and GMNL-33, mean it can make anti-allergy claims as well as oral health claims for products it hopes to launch on the Chinese market this year.
These would most likely be in the form of probiotic yoghurt drinks or infant formula milk powders. The company is in negotiations with major Chinese dairies but said negotiations had been stalled to a degree by the melamine infant formula crisis that erupted on the Sino peninsula last year.
Chinese approval
But in China, under its Management Method for Food for New Resources, the strains have only been approved for foods, not supplements.
“The Chinese have now approved 15 probiotic strains,” GenMont spokesperson David Silver told NutraIngredients.com this morning. “But these are the only ones relating to anti-allergy and bad breath benefits.”
The company said phase-2, clinical trials backed the benefits of the allergy strain – GM-080, although NutraIngredients.com was unable to determine whether these studies had been peer-reviewed at the time of publication. Twelve papers involving its products had been published, the company said.
Anti-allergy
The company said GM-080 could reduce allergic symptoms by 40-50 per cent after a month of consumption and the risk of allergy recurrences after six.
Those who suffered from the likes of asthma, allergic rhinitis, ectopic dermatitis, allergic conjunctivitis, urticaria and food allergies were the target market as well as well as seasonal allergy patients and expectant mothers.
Its other probiotics include products marketed on anti-arthritis, and cholesterol lowering potential.
FAAN News
This is a great group and an unparalleled resource for anyone that cares for people or children with allergies.
Beneficial bacteria reduces eczema - an allergy indicator
This was another article posted in "dairy news"...though the supplements in the test were not a food item, probiotics are often added to dairy. There are alternative non dairy sources for those with an actual dairy allergy. Good Belly has a line of probiotic juices. My favorite is the Watermelon Pomegranate.
Probiotics may reduce eczema risk by 60 per cent
The study, published in the European Journal of Allergy and Clinical Immunology, used Winclove Bio Industries’ Ecologic Panda mixture containing one billion colony forming units (CFU) of each Bifidobacterium bifidum W23, Bifidobacterium lactis W52 (previously classified as Bifidobacterium infantis), and Lactococcus lactis W58.
Eczema, also known as atopic dermatitis (AD), is one of the first signs of allergy during the early days of life and is said to be due to delayed development of the immune system. According to the American Academy of Dermatologists it affects between 10 to 20 per cent of all infants, but almost half of these kids will 'grow out' of eczema between the ages of five and 15.
Study details
Researchers from University Medical Center in Utrecht, Wageningen University, and Sint Antonius Hospital in Nieuwegein, recruited 157 pregnant women and randomly assigned them to receive the probiotic mixture, or placebo, for the last two weeks of pregnancy. The infants subsequently received the supplements for their first year of life.
The Dutch researchers report that parental-reported eczema was 58 per cent lower in the intervention group compared with placebo during the first three months of life, after which the incidence of eczema was similar between the groups.
Furthermore, faecal samples from the infants showed a higher colonisaiton by Lc. Lactis, they added.
In vitro test showed that production of IL-5, one of the protein-like cytokines produced by white blood cells called T-helper type 2 (Th2) lymphocytes during the immune response to allergy, was significantly lower in the probiotic-group compared with the placebo-group.
“The results of this study suggest that primary prevention of eczema by perinatal administration of probiotic bacteria indeed involves modulation of the early colonization of the intestinal microbiota, which may result in modulating the development and maturation of the infants’ immune system,” wrote the researchers.
“Modulation of the immune response via interaction with intestinal dendritic cells with subsequent effects on T-cell differentiation and induction of regulatory T cells has been suggested,” they added.
According the FAO/WHO, probiotics are defined as "live microorganisms which when administered in adequate amounts confer a health benefit on the host".
Source: Allergy
Published online ahead of print, doi: 10.1111/j.1398-9995.2009.02021.x
“The effects of selected probiotic strains on the development of eczema (the PandA study)”
Authors: L. Niers, R. Martín, G. Rijkers, F. Sengers, H. Timmerman, N. van Uden, H. Smidt, J. Kimpen, M. Hoekstra
To me what this seems to indicate is that the further we pull away from our environment, the more harm we cause ourselves in our attempt to stand apart from this symbiosis somehow.
As part of our effort to "clean up" and industrialize the food supply we've moved from our local methods of preserving and pickling... which naturally created probiotic foods that were eaten frequently. Though to some extent we've obtained a somewhat sterile food supply with pasteurization and irradiation, the fact is we're also killing the bacteria that allowed us to achieve a balance with the environment.
Naturopaths and raw foodists pass on an oral tradition of the benefit of probiotics and the enzymes found on raw food. It's gratifying to see a scientific study that validates this to some extent. It's both interesting and sad to me that the dairy industry is a main player in valadating/promoting this discovery. I hope this study brings forth many that further examines the mechanism of this phenomenon.
I've been told 80 - 85% of our immune system is in our intestines where these beneficial bacterias reside. When our digestive systems functionally break down we lose our ability to heal and fight disease.
Negotiating allergies
Thought this was an interesting article by a parent about how to evaluate and reevaluate his child's food allergies.
French toast
Hey... Someone here at work gave me that same link.
Apparently sometimes this guy has a series of articles on this and posts his recipes every now and then too.
Successful treatment of peanut allergies
Treating children with peanut allergies by giving them increasing, but miniscule (the article warns, don't try this at home) amounts of peanuts over several years has resulted in children with increased tolerance and 5 children seeming to be allergy free.
Asthma and Allergies are linked
Both asthma and allergies seem to be the bodies response to removing pollutants from our system.
Though we may find a pill for one symptom for one genetic type now, what's going to happen when we have to find the right pill to take for breakfast?
I posted an article here on the Asthma page talking about the increase in allergies. It's now being addressed by the British because it's finally hitting them in the pocketbook. America will get there too, if you want my opinion.
My son may be the extreme case right now that the FDA literally doesn't have to consider with their safety regulations, but it won't be so easy to ignore when the law of averages catches up.
In any case, this is part of my motivation to buy organic food whenever possible.
100% Milk fabric
100% Milk fabric
I wonder how this really could be considered "hypoallergenic" when it's manufactured out of a known allergen. *SIGH*
Here's another article on the product.
I'm completely frustrated and annoyed.
The only way to be 100% safe from dairy if you have an allergy is to make your own food from scratch (or anything you put in your mouth such as gum), create your own pharmaceuticals (including and especially antihistamines and asthma prophylactic meds), lotions, creams and soaps, make your own paint... and apparently make the fibers of and weave your clothes too.
Human Pollution
Here's an article I stumbled across that I find very informative. It's written about women's health but contains information pertinent to all.
I seriously believe that these things play a huge part in the development of allergies and asthma today. I don't think that it's chance that endocrine disruptors are only recently being studied - and that researchers still have not found the major influential cause of food allergies or the exponential rise of a myriad of diseases since the industrial revolution.
Endocrine Disruptors
An endocrine disruptor is a synthetic compound that mimics a natural hormone when it is absorbed by the body. It can turn on, turn off, or change normal signals. It can have the effect of altering normal hormone levels, triggering excessive action, or completely blocking a natural response. Any other bodily function controlled by hormones can also be affected.
A CDC report from July 2005 found that the bodies of Americans of all ages contain an average of 148 synthetic chemicals.
Some humans seem to be better at dealing with these substances, but we suspect that the increase in chemical and medical sensitivities, childhood cancers, infertility rates, learning disabilities, autism and mood disorders may relate in some way to the sea of endocrine disruptors in which we all swim.
***So that part got me wondering about my recent soy dilemma too.****
Much research has shown that phytoestrogens, such as those found in soy, are not disruptive to the natural workings of the endocrine system. The reason behind this is that the human body has co-evolved over time with plants and generally moderates the impact of phytoestrogens through an adaptogenic response. Some plant estrogens are naturally neutralized, others are easily excreted, and most do not accumulate in body tissue (unlike synthetic compounds and heavy metals). The half-life of a phytoestrogen is measured in minutes, while the half-life of various synthetic compounds, like DDT, may be years or even decades.
***This actually made sense to me for a lot of reasons... even so I'm thinking the endocrine disruptors may be what prevents the body from processing phytoestrogens the way in which the body has evolved to. It's like a toxic overload. This whole cooevolution issue again reinforces my concern about our genetically modified food supply. I only skimmed the article, but I don't think they expressly talked about this concern.**
They have a list of endocrine disruptors in the article and a list of things you can do to avoid them.
More talk about this sort of thing on the Plastics forum too.
Food Allergies
I'm allergic to walnuts, pecans, and brazil nuts. I'm also allergic to raw apples. My lips begin to swell up, my throat starts to close up on me and it's hard to swallow. I hear that every reaction gets worse each time, so I'm afraid what would happen the next time I eat one of these things. I have an epi-pen just in case I need it.
What's weird is that I can eat apple pie. I'm not sure why. I guess whatever it is I'm allergic to is cooked out of the apple... weird.
Chiro
I had a friend that was convinced that he had his peanut allergy cured from going to the chiropractor.
This wouldn't surprise me...our middle daughter had to be rushed to the emergency room when she was one and a half and put on a nebulizer because of what was diagnosed as asthma.
After it happened we consulted a chiropractor we used that practices TBM and he told us to call him if she has another attack. So, sure enough a week later we are at his office now. He treats her and gets her breathing right again within minutes without a nebulizer and after a few weeks treatments, and the purchase of a home nebulizer just in case, she has been attack free ever since.
Since then we've all been treated for various allergies...our oldest daughter also had a similar reaction once to garden variety weed control chemical that we used on our lawn and he treated that with similar results. We still use the same chemical today we did then and she has no problems at all.
I don't know this will cure peanut allergies. We haven't had that, but it's something to consider. We've seen unquestionable results from TBM and this is with things like the flu as well where it's unmistakably results.
Scary Stuff
This is really scary stuff, I think. I know I'm lactose-intolerant and I'm starting to think I might have a gluten allergy as well. The lactose-intolerance is relatively new; in high school I would easily consume three gallons of milk in an average week. Now, one cup makes for a bad night. The other allergy that might be gluten is also pretty new. Gluten allergies are bad because they're progressive. I love making and eating breads and pizza, so if I have a gluten allergy I'm more or less willing to deal with the side effects. Unfortunately, if I do that, then my condition will probably turn into stomach or colon cancer.
I'm willing to do whatever it takes to keep my child out of this dilemma. My wife isn't what I'd call a generally healthy person, it seems like she's sick all the time. However, she has no known food allergies. Her mom followed similar dietary restrictions that we are taking with our daughter, although we have more information so we're even stricter. Since we so far have a control group of one, and the results have taken so long to be noticeable in me, we won't know for years and even then it won't be all that conclusive (unless it's conclusively negative that strict monitoring of diet made no difference).
Small Rays of Hope...
The main allergy researched is peanut because it is usually the most lethal and long lasting. I can only hope that anything they find in relation to this can be used for any food allergy - including milk.
Some peanut allergy studies:
Though the allergy can last a lifetime, another 2003 study indicates that 23.3% of children will outgrow a peanut allergy.[16]
Peanut allergy has been associated with the use of skin preparations containing peanut oil among children, but the evidence is not regarded as conclusive.[17] Peanut allergies have also been associated with family history and intake of soy products.[18]
Canadian researchers are part of an international team that claims to have found clues that could identify those at risk of fatal peanut allergies.
The study found that nine people who died of peanut allergies had significantly lower levels of an enzyme which normally breaks down the chemical that causes bronchial spasms.
They also had high levels of IgE antibodies that are activated during an allergic reaction.
There is no concrete evidence to explain the rising number of peanut allergies in industrialized countries over the past decade.
Some children who are allergic to peanut may actually outgrow the life-threatening allergy, some American researchers have reported.
"Although we once thought peanut allergy was a life-long problem, we now believe certain children, namely those with low levels of allergy antibodies, may outgrow it," Dr. Robert Wood, pediatric allergist and immunologist at Johns Hopkins Children's Center in Baltimore, said Thursday.
Rich Man's Disease
You'll find a link below this text to an article I think you should read. I need to say in advance that it is not focused on the subject matter here, although I think it's a really good article to read. The relevant portion:
What about peanut allergies?
"We just don't see it," Shepherd says. "In developing countries food allergy
is not nearly the problem that it is in industrialized countries.
Context: Malnourished children are being given a food that is essentially peanut butter with added nutrients.
This makes me wonder what studies will be kicked off by revelations like this. The whole "Back to Sleep" campaign was in a studied response to the question of why children in third world countries are less likely to die of SIDS than children in the US. The two biggest differences found were that children in third world countries sleep on their backs and with their parents. Overcoming the co-sleeping taboo in the US is still proving to be an issue, but getting parents to put their children to sleep on their backs wasn't too tough. It has made a noticeable impact on the frequency of SIDS. But, then, no one was all that invested in the concept of having children sleep on their stomachs, either. Just seeing how formula companies try to squelch all indicators of the superiority of breastfeeding and downplay what does get out might be a good indicator of what all the industries getting rich off keeping Americans fat and sick might do when their cash cow [pun intended] is threatened.
co-sleeping
ESi,
I'd be interested in what you've read on co-sleeping preventing SIDS as when I was practicing it I heard very much the opposite claim from many. Thanks.
More Info
I talked it over with my wife and got some more of the skinny. No study has been published with any believable indication that co-sleeping causes SIDS. The problem here is that because no one knows exactly what causes SIDS, it's still means "death by undetermined causes". So, what has happened is that some infants have died while co-sleeping, and because no one could be certain exactly what killed them, the cause of death was listed as undetermined, and other people called it SIDS.
Also, people co-sleep for various reasons. In our family, it's because we desire that powerful emotional bond it gives us and because we feel that there are definite psychological and physical benefits to co-sleeping. However, some parents co-sleep simply because they can't afford a crib or otherwise have no other place for their baby to sleep. These parents are unlikely to have attained the knowledge of creating a safe environment for co-sleeping, since that big stack of literature the hospital sends you home with doesn't cover it at all and lots of people just don't think there might be more to it than just tossing the baby in the bed. Just imagine a mother who has a glass of wine and a couple of cigarettes before curling up with her baby, then wakes up the next day with a dead child. SIDS might get the blame, and at one point in the not-so-distant past it almost definitely would have.
Because the cause of SIDS is undetermined, nothing can be considered a "cure" for SIDS. For instance, doctors know for a fact that a woman who consumes 80mg per day of protein throughout her pregnancy absolutely will not suffer pre-eclampsia. However, they don't call it a "cure" for pre-eclampsia because the true cause of pre-eclampsia isn't known, and consuming less than that amount of protein doesn't mean a woman will suffer from pre-eclampsia. It's kind of the same thing with co-sleeping and SIDS. Just like there are theories on what causes SIDS, there is a theory on how co-sleeping might help prevent SIDS. Because the breathing response is not triggered by lack of oxygen but by build-up of carbon dioxide, adding the mother's carbon dioxide to the mix of air the infant is subjected to might encourage the infant's system to continue breathing. Also, as I said in my other post, the infant's regulatory systems do appear to take cues from the mother's.
co-sleeping
I'll have my wife get out whatever she found. She did all the real hard work on this one.
Basically, there's no proof, and I'm not sure an in-depth study has been done. It started with a basic question: Why do infants in the US die of SIDS more often than infants in undeveloped countries? The two factors they found different were that infants in those countries sleep on their backs and sleep with their parents. Americans have been told to put their babies to sleep on their backs for a while now, and the instances of SIDS have been reduced dramatically. The instances of SIDS deaths in this country is still higher than in those undeveloped countries however, but that's not proof that co-sleeping would make up the remainder of the difference. There are still other factors that may contribute to SIDS, such as smoking, general environment, undetected allergies that affect the respiratory system, and other things that could distinguish the average American baby from a baby in a third world country.
I don't know of anyone who has ever had a compelling reason to say that co-sleeping causes SIDS. Unsafe practices during co-sleeping has caused infants to die, but I think ruling those deaths as being caused by SIDS was to protect well-meaning, traumatized parents who unintentionally smothered their children from being charged with involuntary manslaughter or negligent homicide or something of the like. There's a fear that a mother will roll over on the baby, which has pretty much zero chance of happening except with very heavy women. Mother and child have a special bond and something in that prevents Mom from ever doing such a thing. It's not a proven thing either, I guess, but in countries where co-sleeping is the norm, they don't have the slightest concern over it and have the statistics to back them up. Unfortunately, only Mom and baby have that bond; Dad and anyone else who winds up in the bed need to be shielded from hurting the baby. Extreme caution must be taken to ensure that the fitted sheet is very tight, and all cover sheets, pillows, and blankets must be kept away from baby. These precautions only cover the smothering hazards of co-sleeping. As you well know, there are a host of other things that have to be considered for safe co-sleeping. I'd imagine that a lot of the anti-co-sleeping attitude is just a general distrust of people to know what the right thing to do is or to actually do what they know the right thing is.
I won't say that co-sleeping prevents SIDS, although it is my opinion that it does at least contribute to its prevention. For one thing, Mom wakes up whenever she senses something isn't right with baby. A lot of times, SIDS seems to happen when a baby's system malfunctions and just stops sending the command to breathe too long. Any sort of stimulation seems to reset that system. When Mom 'senses' something isn't right and wakes up to check the baby, that alone could be enough to get the baby going again. Also, being in such close proximity to Mom also means that baby's system has a perfectly functioning one to emulate. It has been shown that baby's heartbeat will come in tune (not necessarily sync'ed) with Mom's during co-sleeping, so it seems rational that other functions, such as breathing, might take cues from Mom as well.
Politics involving the minority
Unfortunately, this "Rich Man's Disease" isn't prevalent enough for most people to care. Here are a few articles scratching the surface of that:
Allergic children may break out in hives if they so much as kiss someone who has eaten a peanut or if they merely touch a surface -- like a table or doorknob -- where peanuts or peanut butter have been. Peanut protein survives for six months.
Not only is the peanut protein long-lived, but the peanut allergy can survive death. A man who received a liver and kidney transplant inherited a peanut allergy from the donor, a 22 year-old who had fallen into a coma and died after eating satay sauce.
Several months after the transplant, the recipient suffered a skin rash and difficulty breathing after eating peanuts. Doctors traced the newly developed allergy to blood cells primed to recognize the allergins that were passed along with the liver.
One illustrative anecdote involves a peanut-free day care center where an aide spotted a peanut butter and jelly sandwich in the hands of a 4-year-old. Turns out the mother had been making her child's sandwiches with a 2-inch circle of peanut butter in the middle, then surrounding it with jelly to hide the peanut butter.
When told her child could not remain in the program if she didn't abide by the school's rules, she replied that her child's rights were being subverted by "this peanut allergy nonsense."
Unfortunately, parents with such attitudes can convey them to their children, thus fostering a ten dency to tease the allergic child. In another few years, the smiles and compliance that Ezra encounters today when he announces his allergy may change to peer scoffing, teasing and harassment ("Ha, ha, you're the peanut kid!").
**This happened to my son in daycare. He was called "allergy boy" and beaten up by a group of kids regularly. He told me about the teasing once. I talked to the daycare staff and they told me they would handle it. They handled it by threatening my son and telling him "not to go crying home to mommy" everytime someone said something. He stopped telling me about it.
Exaggeration? Tell that to the 7-year-old St. Louis girl who had to be hospitalized for two days after having crumbled peanut butter crackers dumped on her head by a boy on her school bus who was harassing her about her allergy.
**The culmination of the teasing at this daycare resulted in the main bully splashing my son in the face with milk from his breakfast cereal. My son had to be taken to the ER. This kid saw my son's face - it looked like it'd been whipped by a cat-o-nine tails. I lectured him - the bully cried - and 2 days later gave my son a bloody nose on the playground. There was no remorse.
Banning peanuts from schools....
RUSH LIMBAUGH: But isn't this a typical liberal solution? You've got 440 students and 1 of them has a peanut allergy. What do you do? You ban peanut butter from all 440 of the children, spread the misery around, deny everybody the right to eat peanut butter in order to protect the 1 child who cannot.
LEE HOCHBERG: Some parents are angered by accommodations made for other allergies. When a preschool in Virginia banned milk, cheese, and yogurt to protect a student with an allergy to dairy products, some parents pulled children from the school and withheld tuition. Critics also have lashed out at a recent ruling from the U.S. Justice Department that obligates school teachers to treat allergic reactions. Peanut-allergic children could need a life-saving shot of adrenaline but La Petite Academy, the nation's second largest child care provider, said it would not take on the liability of administering it. Parents of allergic kids said the school was violating the Americans with Disabilities Act, which requires schools make reasonable accommodations to people with disabilities. In an October settlement with the government the Academy agreed to administer adrenaline. Like many schools, St. Ann's Episcopal in Denver, has begun adapting to its peanut-allergic children, like nine-year-old Caroline Curtiss. Though Curtiss brings her own peanut-free lunch, the school stopped using peanut oil in its cafeteria food to reduce her risk of reaction.
Though most teachers at this training session seemed to embrace their new duty, one wondered why she was suddenly being asked to practice medicine.
PEGGY JOHNSON, State Representative, (R): I think that it's going on beyond what would be expected of any teacher. A teacher is trained to teach. They expect to walk in the classroom and teach. They do not expect to be technicians, medical technicians, or health care providers. To assume the responsibility for the total well-being of the child in the classroom, I don't think so.
The chairwoman of Washington State's House Education Committee, Johnson says teachers already have enough to do as educators without becoming, as she puts it, social workers.
Food Allergy Network: This is a medication that will save someone's life. It's the only medication that will turn around an allergic reaction. How can we honestly sit there and say, I don't know if I want to do that?
How to handle allergies in the school system
I am again faced with this as the school year starts and I have the myraid of papers to present to a new school in order to help my child survive the year.
This somewhat political debate is still ongoing...
I'm constantly amazed at the lengths some parents will go to in order to insure that they don't have to deal with other family's problems within their community.
(Article highlights below)
Peanut Butter Bans
More schools than ever are banning peanuts and peanut products as the number of kids diagnosed with the potentially life-threatening allergy has climbed dramatically in recent years.
The main worry for Searles, like many parents, is that her son would have a reaction without actually eating a peanut product. It's possible for Matthew to have a reaction from touching a table or utensil with peanut butter on it and then putting his hand into his mouth or rubbing his eyes, Searles said.
It’s a legitimate concern, Sampson said. That’s why he supports peanut bans in preschools and kindergarten classes, where students are prone to putting their hands in their mouths. As children grow older, he favors carefully cleaned peanut-free tables in the cafeteria, hand washing and other common sense precautions.
“As children get older and more responsible, you don’t have to have anything like a ban,” he said. “You want them to learn to deal with the situation.”
“People were extremely rude,” she said. “They just thought it was a ridiculous request.”
People left nasty posts on local message boards. One online writer suggested ending the issue by putting all the allergic children in a room together and feeding them peanuts, Searles said.
Alternative to food bans
Parents opposed to the bans have an unlikely ally — an advocacy group for people with food allergies. The Food Allergy & Anaphylaxis Network in Fairfax, Va., recommends schools treat each student's allergy individually and adopt plans that emphasize continued vigilance rather than food bans.
The statistics today
It is, said TODAY medical editor Dr. Nancy Snyderman, a growing problem in the United States and in other developed nations. An estimated 12 million Americans suffer from food allergies, with 6.9 million allergic to seafood and 3.3 million allergic to peanuts or tree nuts. Eight foods — milk, eggs, nuts, wheat, soy, fish and shellfish — account for 90 percent of food allergies.
While doctors try to figure out the reasons for the rise, the situation pits parents against each other and puts school districts in the middle. Between 1997 and 2002, the rates of peanut allergies in children under age five doubled, said Dr. Hugh A. Sampson, president of the American Academy of Allergy, Asthma & Immunology.
I wonder what I can do in order to make things easier for other allergic children down the road. My son can't eat school lunch - the district won't even try to feed him unless he's declared disabled (no matter what the doctor or his blood work says). Years ago he qualified for disability but this was based upon him being sent to the ER on a regular basis. When we had his allergies in control with medication he was determined ineligible. I was told with an attorney I could get this status reinstated because it was an area where the law was vague, but I wondered "why should I do this" when I wasn't confident that going through all this effort just to qualify for a school hot lunch would have resulted in a meal that wouldn't kill my boy in the first place. And now... They recently scaled back disability laws -- it's hard to even think there will be legal recourse for families that carry this kind of future. I almost feel like the government is saying "let's lock them all in a room..."
All my ranting aside... I agree with the The Food Allergy & Anaphylaxis Network . A ban would be creating a whole host of problems of its own. For instance - peanut butter is one of the few things my son actually can eat without dying. How do you determine which child has more of a right to live? - You just can't. Should these children be forced to attend home schooling because the government/community does not want to have to deal with it? Even if such a thing could be funded (tell me how single parents could do this and keep a job that would support a family) - this might work only until the allergy rates are keeping everyone at home.
This is something that could be taken care of by people just paying attention to what is going on and acting in a responsible manner. But beyond that... the health care system in this country is really getting scary out of control. We need to focus on preventative medicine in order to find out what's causing this phenomenon. Here's the Catch 22 - the pharmeceutical companies make their profit and living off of our ailments and have a vested interest in keeping us "just sick enough"... but if they don't make a profit off of our ailments, where will the cutting edge medical research come from? How do we find an altruistic company? If the government got involved - what could they really do except narrow and restrict the power to people on their payroll that aren't necessarily any less corrupt? I don't even know what the answer is.
attitudes toward people with allergies
Crazy
MOD: Duplicate as anonymous again... sorry :(
I can't believe people are getting so crazy about this. The peanut allergy, among others, is extremely deadly, but the treatment is excessively simple. Teachers are required to know CPR, and I'm fairly certain they have to occasionally update that training. Most people with these intense allergies are well aware that they have them, including the kids, so they carry around one of those little pen things that they can use in an emergency. Those are WAY easier to use than administering CPR is. I'm generally sensitive to the issues that America's teachers are confronted with, but teachers who whine about having to deal with this sort of thing remind me of those people who sign up for the military and then complain because they have to do military things. Whether they like it or not, being a teacher in an American school also means they are daycare providers, with all the responsibilities that go along with that, and whining about those base expectancies earns no sympathy because they had the freedom to choose the line of work they went into.
Roasting vs Boiling
This is one of the things they are considering. Potentially the way we process peanuts may cause the complication. Here are excerpts from 2 different articles:
Roasting enhances allergic properties in peanuts Another new study found that roasting may play an important role in enhancing the allergenic properties of peanuts. Soheila J. Maleki, PhD, from the United States Department of Agriculture discovered that roasting causes the major peanut allergen, Ara h2 to become a stronger digestive enzyme inhibitor and more resistant to digestion. The study is the first to show that roasting alters the functional characteristics that directly influence the allergen's allergic potential. These findings have important implications for studies of other allergens, where research has previously focused primarily on structural characteristics. This may explain why peanuts are such a potent allergen.
A theory of the development of peanut allergy has to do with the way that peanuts are processed in North America versus other countries like China and India. Peanuts are widely eaten in China and India but peanut allergies are almost unheard of there. According to a 2003 study, roasting peanuts (vs. boiling), as more commonly done in North America, causes the major peanut allergen Ara h2 to become a stronger inhibitor of the digestive enzyme trypsin, making it more resistant to digestion.[15] Additionally, this allergen has also been shown to protect Ara h1, another major peanut allergen, from digestion - a characteristic further enhanced by roasting.[15]
Interesting
That will be interesting to see where this goes. I can see where it would make an impact, but you have to wonder how widespread it will be. Are peanuts that wind up as oil generally roasted or boiled first?
What I'd like to see done are conclusive studies that follow the age food is introduced to a child and food type. For instance, our daughter was exclusively breastfed until 6-1/2 months of age. I was thinking about pushing until 7 but it started to seem that my wife's supply couldn't keep up with our daughter's demand. We began introducing solids slowly, and are gradually see-sawing from breastfed as primary nutrition with solids as supplements to solids as primary with breast milk as a supplement. Foods were introduced based on how allergenic they are known to be. We started with avocados, then went to rice, then bananas, and then we opened it up to things like sweet potatoes. We are absolutely not allowing her to have any cow's milk until she is over a year old (I'd have to check our calendar for when it's OK). I think our schedule prohibits all nuts until she's over two years old. Our gamble is that she'll never have any food allergies of any kind, and, as I illustrated in another post, she may also have a dramatically reduced likelihood of non-food related allergies as well.
The thing is, I certainly want my daughter to experience all the wonderful things in life, including good food. I'm a peanut butter fanatic myself, and I certainly don't want to deprive her of it. That said, I know for a fact that a human being can live out a full, happy, healthy life without ever tasting peanut butter. So where is the harm in keeping it away from her for a couple of years? I don't see any. Some of the other allergenic foods are harder to keep her away from just because they are so prevalent: wheat, cow's milk, etc. exist in so many foods targeted for infants that I'm sure someone somewhere is cooking up a conspiracy theory over it. I was in the infant's section of our local grocery store last week and saw a box of zwieback crackers, which I remember as being a big hit from experiences around other babies. With my recently trained eye, I scanned the ingredients list and almost went into cardiac arrest on the spot. I can't believe that the big-name company who made them would pack so many highly allergenic foods into a cracker intended for kids less than a year old! But, there it is.
Tangent on Cow's Milk
Can you imagine what it would be like...
To be afraid of every bite of food that you eat because it quite literally may kill you?
To wonder if your boyfriend/girlfriend ate something you are allergic to - because if you kiss them you might end up dying???
To wonder if you're going to kill your child who has allergies by purchasing the wrong packaged food knowing that the safety levels considered by the government for labelling laws do not address highly allergic people?

Labelling concerns
Food label warnings about what the product may contain are possibly missing the risk of cross contamination.
"“Milk protein concentrations in unlabelled dark chocolate sprinkles, probably present as a result of cross-contact, were shown to reach levels that can cause serious allergic reactions within the milk-allergic population,” wrote the researchers in the journal Food Additives and Contaminants."
"“Now and in the future probabilistic allergen risk assessment is a way to calculate the health risk impact of contaminated products and to provide a sound basis for risk-management decisions,” they wrote"
Allergen cross-contamination raises labelling concerns